In sepsis-associated cardiotoxicity, remifentanil reduces oxidative stress, inflammation, and apoptosis to maintain mitochondrial biogenesis by inhibiting NFkB and cas-3 immunoexpressions and enhancing SIRT1 upregulation.
Immunopharmacology and immunotoxicology, cilt.48, sa.1, ss.1-10, 2026 (SCI-Expanded, Scopus)
- Yayın Türü: Makale / Tam Makale
- Cilt numarası: 48 Sayı: 1
- Basım Tarihi: 2026
- Doi Numarası: 10.1080/08923973.2025.2585087
- Dergi Adı: Immunopharmacology and immunotoxicology
- Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus, BIOSIS, EMBASE, MEDLINE
- Sayfa Sayıları: ss.1-10
- Anahtar Kelimeler: apoptosis, cardiotoxicity, oxidative stress, remifentanil, Sepsis
- Süleyman Demirel Üniversitesi Adresli: Evet