Is Carbon Monoxide Poisoning a Potential Trigger for Spontaneous Coronary Artery Dissection? A Case Report and Review of the Literature Karbonmonoksit Zehirlenmesi Spontan Koroner Arter Diseksiyonunu Tetikleyebilir mi? Olgu Sunumu ve Literatür Derlemesi
Turk Kardiyoloji Dernegi Arsivi, cilt.54, sa.7, ss.616-621, 2026 (ESCI, Scopus, TRDizin)
- Yayın Türü: Makale / Tam Makale
- Cilt numarası: 54 Sayı: 7
- Basım Tarihi: 2026
- Doi Numarası: 10.5543/tkda.2025.11859
- Dergi Adı: Turk Kardiyoloji Dernegi Arsivi
- Derginin Tarandığı İndeksler: Emerging Sources Citation Index (ESCI), Scopus, Central & Eastern European Academic Source (CEEAS), EMBASE, Directory of Open Access Journals, TR DİZİN (ULAKBİM), Biomedical Reference Collection: Corporate Edition (EBSCO), Health Research Premium Collection (ProQuest)
- Sayfa Sayıları: ss.616-621
- Anahtar Kelimeler: Acute coronary syndrome, carbon monoxide poisoning, spontaneous coronary artery dissection
- Açık Arşiv Koleksiyonu: AVESİS Açık Erişim Koleksiyonu
- Süleyman Demirel Üniversitesi Adresli: Evet
Özet
Spontaneous coronary artery dissection (SCAD) is a rare but potentially fatal cause of acute coronary syndrome (ACS), often associated with younger individuals and women without traditional cardiovascular risk factors. Carbon monoxide (CO) poisoning, a known cause of systemic hypoxia, has been implicated in various cardiovascular complications; however, its role in triggering SCAD remains scarcely reported. We present the case of a 55-year-old woman who developed SCAD following acute CO poisoning caused by a domestic gas leak. The patient presented with altered mental status and respiratory failure, requiring hyperbaric oxygen therapy. Following stabilization, typical anginal symptoms emerged, accompanied by rising cardiac biomarkers suggestive of myocardial injury. Coronary angiography revealed spontaneous dissections in both the distal left anterior descending artery and the distal right coronary artery without critical flow limitation. The patient was managed conservatively with medical therapy and remained asymptomatic at six-month follow-up. This case highlights the importance of considering SCAD in patients presenting with cardiac symptoms following CO poisoning and underscores the need for vigilance in atypical presentations of myocardial injury.